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Topic: Allergic encephalomyelitis

	Encephalomyelitis - acute disseminated encephalomyelitis
		Encephalomyelitis encephalomyelitis definition is a general term for inflammation of the brain and spinal cord, experimental autoimmune encephalomyelitis model describing a number of disorders:
		acute allergic encephalomyelitis equine encephalomyelitis disseminated canine acute disseminated encephalomyelitis encephalomyelitis or postinfectious encephalomyelitis, a demyelinating disease of the brain demyelinating viral encephalomyelitis and spinal cord, possibly triggered by vaccination or viral infection;
		myalgic encephalomyelitis allergy and acute disseminated encephalomyelitis myalgia encephalomyelitis acute disseminated encephalomyelitis and pediatric neurology encephalomyelitis or chronic encephalomyelitis symptoms fatigue syndrome, a debilitating syndrome of unknown acute disseminated encephalomyelitis etiology which was once thought myalgic encephalomyelitis to involve inflammation of herpes virus myalgic encephalomyelitis the brain.
	www.medicalgeo.com /Med-Diseases-E/Encephalomyelitis.html (225 words)

	[No title]
		Experimental allergic encephalomyelitis is an intensely aggressive inflammatory disorder in which myelin loss occurs, predominantly as a bystander reaction, in a small perivascular sleeve of tissue as opposed to MS which is characterised by large demyelinating plaques, conspicuous astrocyte proliferation with or without scant lymphocytic infiltration.
		Experimental allergic encephalomyelitis has been used as a model for MS and it is the best studied organ-specific autoimmune disease: it is not acceptable as a model for MS for many reasons, some of which have already been alluded to.
		The neurological unity of acute haemorrhagic leukoencephalitis and acute disseminated encephalomyelitis.
	www.mult-sclerosis.org /news/Nov2002/FullTextofWrongDiseaseTheory.html (15689 words)

	Raymond A. Sobel, M.D. - Faculty & Researcher Profiles - Stanford School of Medicine
		Sobel RA, Schneeberger EE, Colvin RB "The immunopathology of acute experimental allergic encephalomyelitis.
		Sobel RA, Natale JM, Schneeberger EE "The immunopathology of acute experimental allergic encephalomyelitis.
		Sobel RA, Colvin RB "The immunopathology of experimental allergic encephalomyelitis (EAE).
	www-med.stanford.edu /profiles/frdActionServlet?choiceId=printerprofile&fid=4269 (3585 words)

	Post-vaccinal encephalomyelitis
		This experimental allergic encephalomyelitis is mediated by sensitized lymphocytes rather than serum antibody mechanisms.
		Clinical suppression of experimental allergic encephalomyelitis by muramyl dipeptide "adjuvant".
		Schchelkunov, SN et al, "The Role of Viruses in the Induction of Allergic Encephalomyelitis," Dokl Akad Nauk SSSR, 1990,315(1):252-255.
	www.whale.to /vaccines/encephalomyelitis.html (1010 words)

	Igitur-archive (Site not responding. Last check: 2007-10-30)
		T2-weighted and short tau inversion recovery magnetic resonance imaging were used to assess the effect of a neurotrophic adrenocorticotrophic hormone4-9 analogue [H-Met(O2)-Glu-His-Phe--Lys-Phe-OH] on the volume of lesions in the brains of rats suffering from chronic experimental allergic encephalomyelitis, an animal equivalent of multiple sclerosis.
		Twenty weeks after lesion induction rats with chronic experimental allergic encephalomyelitis were killed for histological analysis, to correlate magnetic resonance imaging findings with morphological changes.
		The improved neurological status and the 84% reduction in the lesion volume in the cerebrum of rats chronic experimental allergic encephalomyelitis point to the potential value of trophic peptides in the development of strategies for limiting the damage caused by central demyelinating lesions in syndromes such as multiple sclerosis.
	igitur-archive.library.uu.nl /med/2005-0622-142215/UUindex.html (293 words)

	Office of Research Services (ORS) - Page Title (Site not responding. Last check: 2007-10-30)
		Inhibition of experimental allergic encephalomyelitis in the Lewis rat by paclitaxel.
		Inhibition of experimental allergic encephalomyelitis with an antibody that recognizes a novel antigen expressed on lymphocytes, endothelial cells, and microglia.
		Dendritic cell-derived nitric oxide is involved in IL-4-induced suppression of experimental allergic encephalomyelitis (EAE) in Lewis rats.
	dvrnet.ors.od.nih.gov /ratcenter/reference.html (4636 words)

	Neuropathology Mini-Course
		Acute disseminated encephalomyelitis and postvaccinal encephalomyelitis are apparently identical entities, sometimes also known as postinfectious encephalomyelitis.
		The demyelination in acute disseminated encephalomyelitis occurs in a perivenular distribution.
		An experimental model of the disease may be provided by experimental allergic encephalomyelitis (EAE), a demyelinating disease produced in animals by immunizing them with neural tissue.
	www.pathology.vcu.edu /WirSelfInst/ADE.html (511 words)

	Oligodendrocyte cell death in pathogenesis of multiple sclerosis: Protection of oligodendrocytes from apoptosis by ... (Site not responding. Last check: 2007-10-30)
		Studies of experimental allergic encephalomyelitis (EAE) in caspase 11-deficient mice show that caspase-mediated death of OLGs is critical to demyelination.
		The role of complement in the pathogenesis of experimental allergic encephalomyelitis.
		Attenuation of experimental allergic encephalomyelitis in complement component 6-deficient rats is associated with reduced complement C9 deposition, P-selectin expression, and cellular infiltrate in spinal cords.
	www.vard.org /jour/06/43/1/cudrici.html (4344 words)

	Topic 6-Demyelinating disease
		Allergic encephalomyelitis and allergic peripheral neuritis, progressive multifocal leukoencephalopy (PML) and the white matter involvement in Subacute Progessive Panencephalitis (SSPE) are other examples of human demyelinating diseases.
		As in post infectious encephalomyelitis, the onset of acute hemorrhagic leucoencephalitis is often preceded by a systemic infection.
		Based in part, upon similarities with experimental allergic encephalitis and the failure to identify a pathogen within involved neural tissues, post infectious encephalomyelitis is presumed to be an "allergic" immune mediated process.
	www.path.sunysb.edu /faculty/woz/NPERESS/webclass6b.htm (563 words)

	allergic, Primary Sclerosing Cholangitis Literature
		Bolton C 1995 Recent advances in the pharmacological control of experimental allergic encephalomyelitis (EAE) and the implications for multiple sclerosis treatment.
		Cookson WO, Moffatt MF 2000 Genetics of asthma and allergic disease.
		Smit JJ, van Loveren H, Hoekstra MO, Nijkamp FP, Bloksma N 2003 Influence of the macrophage bacterial resistance gene, Nramp1 (Slc11a1), on the induction of allergic asthma in the mouse.
	www.psc-literature.org /allergic.htm (10294 words)

	ePrintsUQ - The Pathophysiology of Acute Experimental Allergic Encephalomyelitis in the Rabbit
		ePrintsUQ - The Pathophysiology of Acute Experimental Allergic Encephalomyelitis in the Rabbit
		The Pathophysiology of Acute Experimental Allergic Encephalomyelitis in the Rabbit
		Clinical, histological and electrophysiological studies were performed on rabbits with acute experimental allergic encephalomyelitis (EAE).
	eprint.uq.edu.au /archive/00003041 (321 words)

	Cytokine Gene Therapy in Experimental Allergic Encephalomyelitis by Injection of Plasmid DNA-Cationic Liposome Complex ...
		Induction of chronic relapsing experimental allergic encephalomyelitis in Biozzi mice.
		Therapy of chronic relapsing experimental allergic encephalomyelitis and the role of the blood-brain barrier: elucidation by the action of Brequinar sodium.
		Isolation and characterization of cells infiltrating the spinal cord during the course of chronic relapsing experimental allergic encephalomyelitis in the Biozzi AB/H mouse.
	www.jimmunol.org /cgi/content/full/160/10/5181 (5480 words)

	Dual phase regulation of experimental allergic encephalomyelitis by platelet-activating factor -- Kihara et al. 202 ...
		Role of platelet activating factor in the pathogenesis of active and passive models of experimental allergic encephalomyelitis in the rat.
		IL-6-deficient mice are resistant to the induction of experimental autoimmune encephalomyelitis provoked by myelin oligodendrocyte glycoprotein.
		IL-6-deficient mice are resistant to experimental autoimmune encephalomyelitis: roles of IL-6 in the activation and differentiation of autoreactive T cells.
	www.jem.org /cgi/content/full/202/6/853 (6552 words)

	Characteristics of the T lymphocytes involved in experimental allergic encephalomyelitis.
		Characteristics of the T lymphocytes involved in experimental allergic encephalomyelitis.
		However, the unequivocal heterogeneity of the immune response directed at one of the most important myelin constituents, proteolipid lipoprotein (PLP), which occurs either as a primary or a secondary event during experimental allergic encephalomyelitis (EAE), indicates the complexity of the in vivo situation.
		Intramolecular and intermolecular spreading of antigen specificity during the course of the disease indicates that a TCR directed therapy may not be the choice of intervention in established disease even in individual strains of laboratory animals with restricted heterogeneity of the primary MBP-specific response.
	www.aegis.com /aidsline/1996/feb/M9620556.html (459 words)

	The Wistar Institute :: Ellen Heber-Katz, PhD (Site not responding. Last check: 2007-10-30)
		This basic research is being applied to a variety of human diseases, including meningitis, herpes simplex virus infections, heart disease, and spinal cord tissue damage.
		To study autoimmunity, the Heber-Katz research team uses allergic encephalomyelitis as a model, in which certain proteins associated with the disease have shed light on the pathology of meningitis, another autoimmune disease of the central nervous system.
		Experimental allergic encephalomyelitis is a T cell-mediated autoimmune disease induced by myelin antigens in the central nervous system (CNS).
	www.wistar.upenn.edu /research_facilities/heberkatz/research.htm (1113 words)

	Acute disseminated encephalomyelitis -- Garg 79 (927): 11 -- Postgraduate Medical Journal
		Acute disseminated encephalomyelitis (ADEM) is an acute demyelinating
		Acute disseminated encephalomyelitis (ADEM) is an acute widespread
		Pathogenesis of acute viral encephalitis and postinfectious encephalomyelitis.
	pmj.bmjjournals.com /cgi/content/full/79/927/11 (4208 words)

	ePrintsUQ - Demyelination and Neurological Signs in Experimental Allergic Encephalomyelitis
		Because of the reported absence of demyelination in some animals with neurological signs of experimental allergic encephalomyelitis (EAE), it has been suggested that these signs are not due to demyelination.
		The present study demonstrates that there is ample demyelination in the central nervous system (CNS) and peripheral nervous system (PNS) to account for the neurological signs in rats with myelin basic protein (MBP)-induced acute EAE as well as in rats and rabbits with whole-spinal-cord-induced acute EAE.
		Originally published as Pender, M. (1987) Demyelination and neurological signs in experimental allergic encephalomyelitis, Journal of Neuroimmunology, 15 (1): 11-24.
	eprint.uq.edu.au /archive/00002311 (202 words)

	The immunopathogenesis of multiple sclerosis (Site not responding. Last check: 2007-10-30)
		EAE is an acute or chronic relapsing experimental demyelinating disease that is characterized by focal areas of inflammation and demyelination throughout the CNS.
		Transfer of allergic encephalomyelitis in rats by means of lymph node cells.
		Adoptive transfer of experimental allergic encephalomyelitis in SJL/J mice after in vivo activation of lymphonode cells by myelin basic protein: requirement for Lyt-1+2- T lymphocytes.
	www.vard.org /jour/02/39/2/Prat.htm (8244 words)

	Increased Severity of Experimental Allergic Encephalomyelitis in lyn-/- Mice in the Absence of Elevated Proinflammatory ...
		B-cells are critical to induction of experimental allergic encephalomyelitis by protein but not by a short encephalitogenic peptide.
		Soluble recombinant complement receptor 1 inhibits inflammation and demyelination in antibody-mediated demyelinating experimental allergic encephalomyelitis.
		Antibody-mediated demyelination in experimental allergic encephalomyelitis is independent of complement membrane attack complex formation.
	www.jimmunol.org /cgi/content/full/168/6/3105 (4579 words)

	Involvement of Tissue Plasminogen Activator in Onset and Effector Phases of Experimental Allergic Encephalomyelitis -- ...
		Benveniste E (1997) Role of macrophages/microglia in multiple sclerosis and experimental allergic encephalomyelitis.
		Kennedy KJ, Strieter RM, Kunkel SL, Lukacs NW, Karpus W (1998) Acute and relapsing experimental autoimmune encephalomyelitis are regulated by differential expression of the CC chemokines macrophage inflammatory protein-1a and monocyte chemotactic protein-1.
		Suen W, Bergman C, Hjelmstroem P, Ruddle N (1997) A critical role for lymphotoxin in experimental allergic encephalomyelitis.
	www.jneurosci.org /cgi/content/full/22/24/10781 (5682 words)

	Ewa Chelmicka-Schorr, MD
		Schorr's major research interest is the effect of the sympathetic nervous system on immune responses and on experimental autoimmune diseases: experimental allergic encephalomyelitis (EAE), experimental allergic neuritis (EAN) and experimental allergic myasthenia gravis (EAMG).
		Schorr has also studied the immunoaugmenting and immunosuppressing effects of the immunosuppressive drugs in experimental allergic encephalomyelitis.
		Chelmicka-Schorr E., Wiegmann K., Wollmann R., Arnason BGW, and Kim DH (1998): Immunoaugmenting and immunosuppressing effect of FK 506 on experimental allergic encephalomyelitis in Lewis rats.
	ucneurology.uchicago.edu /About_the_Dept/Faculty/Chelmicka-Schorr/chelmicka-schorr.html (211 words)

	Self-antigen-induced Th2 responses in experimental allergic encephalomyelitis (EAE)-resistant mice. Th2-mediated ...
		Self-antigen-induced Th2 responses in experimental allergic encephalomyelitis (EAE)-resistant mice.
		Immunization of a limited number of rodent strains with central nervous system-derived Ags induces experimental allergic encephalomyelitis (EAE).
		These data are consistent with both the ability of Th2-type cells to suppress autoimmunity and a homeostatic mechanism of T cell regulation based on the cross-regulation of Th1 and Th2 cells in the maintenance of peripheral tolerance.
	www.aegis.com /aidsline/1996/jan/M9610401.html (510 words)

	ePrintsUQ - Apoptosis in the Nervous System in Experimental Allergic Encephalomyelitis
		We report here for the first time the occurrence of apoptosis of cells in the spinal cord in experimental allergic encephalomyelitis (EAE), an autoimmune, T-cell-mediated demyelinating disease.
		They were found in both the white and grey matter of the spinal cord in all 4 forms of this disease.
		Thus apoptotic deletion of myelin/oligodendrocyte-specific lymphocytes in the central nervous system in EAE might explain both the subsidence of inflammation and the acquisition of tolerance in this autoimmune disease.
	eprint.uq.edu.au /archive/00002922 (328 words)

	Susceptibility and Resistance to Experimental Allergic Encephalomyelitis: Relationship with ...
		Susceptibility and Resistance to Experimental Allergic Encephalomyelitis: Relationship with Hypothalamic-Pituitary-Adrenocortical Axis Responsiveness in the Rat -- Stefferl et al.
		Williams RM, Moore MJ 1973 Linkage of susceptibility to experimental allergic encephalomyelitis to the major histocompatibility locus in the rat.
		Villas PA, Dronsfield MJ, Blankenhorn EP 1991 Experimental allergic encephalomyelitis and corticosterone studies in resistant and susceptible rat strains.
	endo.endojournals.org /cgi/content/full/140/11/4932 (5351 words)

	Genetic Analysis of Disease Subtypes and Sexual Dimorphisms in Mouse Experimental Allergic Encephalomyelitis (EAE): ...
		Genetic Analysis of Disease Subtypes and Sexual Dimorphisms in Mouse Experimental Allergic Encephalomyelitis (EAE): Relapsing/Remitting and Monophasic Remitting/Nonrelapsing EAE Are Immunogenetically Distinct -- Butterfield et al.
		Genetic Analysis of Disease Subtypes and Sexual Dimorphisms in Mouse Experimental Allergic Encephalomyelitis (EAE): Relapsing/Remitting and Monophasic Remitting/Nonrelapsing EAE Are Immunogenetically Distinct
		Experimental allergic encephalomyelitis (EAE) is the principal animal
	www.jimmunol.org /cgi/content/abstract/162/5/3096 (1116 words)

	[Frontiers in Bioscience 2, d197-206, May 1, 1997]
		Transforming growth factor-beta (TGF-beta), which is produced by many cell types including macrophages and T cells, also inhibits generation of reactive oxygen and nitrogen intermediates (58,59), especially in synergy with IL-10 (43,57,60), and is hypothesized to play a critical role in resolution of inflammatory lesions in experimental allergic encephalomyelitis (61-63).
		The role of contact-dependent signaling in the development of experimental allergic encephalomyelitis has been shown dramatically using transgenic B10.PL mice expressing the T cell receptor reactive with the encephalitogenic peptide (Ac1-11) of myelin basic protein.
		The studies on experimenal allergic encephalomyelitis to date strongly support critical roles for CD40 and TNF-alpha (CD40-induced?) in the pathogenesis of sclerotic lesions and for TGF-beta in remission of the inflammatory episode.
	www.bioscience.org /1997/v2/d/stout1/htmls/197-206.htm (4912 words)

	University of Chicago Hospitals: Ewa Chelmicka-Schorr, MD
		Her goal is to provide up-to-date diagnostic evaluation and treatment for myasthenia gravis, muscular dystrophy, and other myopathies.
		Schorr's major research interest is the effect of the sympathetic nervous system on immune responses and on experimental autoimmune diseases, such as experimental allergic encephalomyelitis (EAE), experimental allergic neuritis (EAN), and experimental allergic myasthenia gravis (EAMG).
		Schorr has also studied the effects of the immunosuppressive drugs in experimental allergic encephalomyelitis.
	www.uchospitals.edu /physicians/ewa-chelmicka-schorr.html (126 words)

	Shaken Baby Syndrome or Vaccine-Induced Encephalomyelitis?----The Story of Baby Alan
		As the inflammatory cells were described to the court as lymphocytes, this would of necessity have been a viral infection, not bacterial.
		The second mechanism would have been a vaccine-induced encephalomyelitis, of which the pertussis, hepatitis B. and Hemophilus influenza bacillus vaccines would have been prime suspects, either individually or in combination.
		Allergic Sensitization Brought about by Vaccines: The increasing incidence of allergic disorders in western nations is now universally recognized, with every third child in industrialized societies having an allergic disorder.(75) Since this trend coincides with vaccine programs, reports are now appearing which address the question of a possible relation between vaccines and increasing allergies.
	www.whale.to /vaccine/yurko.html (8434 words)

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