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| | Febrile Seizures, Amygdala, Autism |
 | | Additionally, (i) SE-induced amygdala damage in humans often leads to permanent damage within the hippocampus (13-14), thereby suggesting impairments in memory among autistics having SE-induced pathogenesis, and (ii) status epilepticus is associated with reduced cerebellar sizes, particularly among Purkinje cells, where gliosis-like scarring is not always evident (36-38). |
 | | Eyes and faces: Atypical, avoidant eye contact is a trait occurring in a high percentage of autistic children (47-48) and is often reported by parents of some autistic children as one of the earliest traits seen the post-vaccination period (21-22,45), a time some individuals have their first recognized FS (10). |
 | | A relatively small number of single neurons within the primate amygdala are known to be responsive to faces; and these neurons have been identified within the basal-lateral and basal accessory nuclei (49- 50), areas functionally similar to those described in rats and other sub-primates (see discussion in 52), ie, areas damaged by SE (12). |
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