
	Where results make sense

Topic: Cerebral hypoxia

Related Topics

Hypoxia medical

Anemic hypoxia

Histotoxic hypoxia

Hypoxic hypoxia

In the News (Wed 3 Dec 08)

Damian Green

Sumner Redstone

Crimson Tide

Tommy Tuberville

Bill Clinton

Wayne Rooney

Vilasrao Deshmukh

Wake Forest

Grand Slam

Oklahoma State

	Cerebral hypoxia - Wikipedia, the free encyclopedia
		Cerebral hypoxia (the pathological condition is called hypoxic encephalopathy) is to a hypoxic condition in which there is a decrease of oxygen supply to the brain or parts of the brain despite adequate blood flow (compare with cerebral ischemia).
		Symptoms of mild cerebral hypoxia include inattentiveness, poor judgment, memory loss, and a decrease in motor coordination.
		Treatment depends on the underlying cause of the hypoxia, but basic life-support systems have to be put in place: mechanical ventilation to secure the airway; fluids, blood products, or medications to support blood pressure and heart rate; and medications to suppress seizures.
	en.wikipedia.org /wiki/Cerebral_hypoxia (292 words)

	Hypoxia (medical) - Wikipedia, the free encyclopedia
		Hypoxia is a pathological condition in which the body as a whole (generalized hypoxia) or region of the body (tissue hypoxia) is deprived of adequate oxygen supply.
		Hypoxia is often associated with high altitudes, where it is called altitude sickness.
		Cerebral hypoxia in which the brain is deprived of oxygen despite normal blood flow.
	en.wikipedia.org /wiki/Hypoxia_(medical) (294 words)

	Cerebral hypoxia Thrombosis Cerebral hypoxia Medical Reference - MedicineOnline.com - Medical Information (Site not responding. Last check: 2007-11-04)
		The term cerebral hypoxia technically refers to a lack of oxygen supply to the cerebral hemispheres (the outer portion of the brain), but it is more typically used to refer to a lack of oxygen supply to the entire brain.
		Complications of cerebral hypoxia include brain death (prolonged vegetative state) -- basic life functions, such as breathing, blood pressure, sleep/wake cycle, and eye opening may be preserved, but there is no consciousness or response to the environment.
		Cerebral hypoxia is a medical emergency with every moment critical to meaningful survival.
	www.medicineonline.com /references/Diseases_and_Conditions/Cardiovascular_Disorders/Thrombosis/Cerebral_hypoxia (674 words)

	Journal of Cerebral Blood Flow & Metabolism - The Effect of Hyperglycemia on Cerebral Metabolism During ... (Site not responding. Last check: 2007-11-04)
		A tissue specimen (60−100 mg) taken from the distribution of the middle cerebral artery of the cerebral hemisphere ipsilateral to the carotid artery occlusion was powdered under liquid nitrogen and weighed on a microanalytical balance.
		A tissue specimen (75−100 mg) was dissected from the ipsilateral cerebral hemisphere in the distribution of the middle cerebral artery and weighed in a tared 5-ml glass vial.
		That cerebral energy reserves are equally or better maintained during ischemia in hyperglycemic compared to normoglycemic adult animals, despite greater brain damage in the former group, suggests that mechanisms other than or in addition to cerebral lactacidosis or energy failure contribute to the ultimate tissue injury.
	www.nature.com /jcbfm/journal/v16/n5/full/9590124a.html (4815 words)

	Disease - Cerebral hypoxia - Hartford, Connecticut
		This term technically refers to a lack of oxygen supply to the cerebral hemispheres (the outer portion of the brain), but it is more typically used to refer to a lack of oxygen supply to the entire brain.
		Sometimes cooling with blankets (hypothermia) is used because much of the brain damage in hypoxia is caused by heat, and those who suffer the condition (if it is caused by drowning in cold water, for example) in cold temperatures have survived longer than those who were affected in higher temperatures.
		Complications of cerebral hypoxia include brain death or a "prolonged vegetative state" where basic life functions, such as breathing, blood pressure, sleep/wake cycle, and eye opening may be preserved, but there is no consciousness or response to the environment.
	www.saintfranciscare.com /11393.cfm (740 words)

	Cecil Textbook of Medicine : />
		Cerebral hypoxia-ischemia can be divided into focal ischemia, caused by vascular occlusion; global ischemia, from complete cardiovascular failure; and diffuse hypoperfusion-hypoxia, produced by respiratory disease or severely reduced blood pressure.
		Cerebral infarction caused by focal vascular occlusion is characterized by destruction of all cellular elements: neurons, glia, and endothelial cells (pan-necrosis).
		Cerebral autolysis is observed most frequently in brain-dead patients who are preserved on mechanical ventilators for more than a few days; it reflects enzymatic autodigestion of the brain tissue.
	www.merckmedicus.com /ppdocs/us/common/cecils/chapters/439_005.htm (1596 words)

	Table of Contents (Site not responding. Last check: 2007-11-04)
		Global cerebral oxygenation, perfusion pressure, and expired CO tension were continuously monitored in 69 adults with acute severe closed brain trauma.
		Cerebral oxygenation was assessed by continuous fiberoptic monitoring of jugular bulb oxyhemoglobin saturation, in conjunction with continuous monitoring of arterial oxyhemoglobin saturation.
		Global cerebral hypoxia that does not respond promptly to treatment appears to be independently deleterious to neurological function in severely head-injured patients.
	www.thejns-net.org /jns/issues/v79n2/abs/n0790228_r.html (228 words)

	Arizona Cerebral Hypoxia Attorneys \| Scottsdale Birth Injury Lawyers \| Failure to Diagnose \| Sun City Grand AZ
		Cerebral hypoxia occurs as the result of a decrease of oxygen supply to the brain, even with adequate blood flow.
		The inability to concentrate and lack of coordination might result in permanent disability to the victim of cerebral hypoxia, particularly when the victim is an infant of a child.
		If you or someone you love has suffered from cerebral hypoxia as the result of a medical procedure, or you suspect that deprivation of oxygen may have occurred, please call us us at 480-905-9208 or 1-888-905-9208 (toll free) to discuss how we can help with this sensitive area of medical malpractice.
	www.arizona-malpractice.com /PracticeAreas/Cerebral-Hypoxia.asp (432 words)

	SRO: Text Only Paper (Site not responding. Last check: 2007-11-04)
		This may lead to structural and functional cerebral macro and microangiopathies, and the ability of the vessels to dilate in order to increase the blood flow to meet the oxygen demands of the brain is compromised.
		Cerebral oxygenation (a,c) and changes in hemodynamics (b,d) as compared to baseline values in (a,b) a healthy control subject and in (c,d) an OSA subject during breath holding.
		Cerebral oxygenation (a,c) and changes in hemodynamic parameters (b,d) as compared to baseline values in a healthy control subject (a,b) and in an OSA subject (c,d) during diurnal napping.
	www.arabmedmag.com /issue-31-03-2005/pneumology/main04.htm (5102 words)

	The Report of ACOG's Task Force on Neonatal Encephalopathy and Cerebral Palsy has been Published
		In contrast, cerebral palsy is a chronic disability of central nervous system origin characterized by aberrant control of movement and posture, appearing early in life and not as a result of progressive neurologic disease.
		Purely dyskinetic or ataxic cerebral palsy, especially where there is an associated learning difficulty, commonly has a genetic origin and is not caused by intrapartum or peripartum asphyxia.
		Similarly, absent cerebral palsy, neither epilepsy, mental retardation, nor attention-deficit hyperactivity disorder are caused by birth asphyxia.
	www.acog.org /from_home/Misc/neonatalEncephalopathy.cfm (970 words)

	Journal of Trauma Lead Abstract (Site not responding. Last check: 2007-11-04)
		Thirty-five episodes of regional cerebral hypoxia were identified in 14 patients.
		Compared with episodes of acceptable cerebral oxygenation, episodes of cerebral hypoxia were noted to be associated with a significantly lower mean Pao2 (144 +/- 14 vs. 165 +/- 8; p < 0.01) and higher mean PEEP (8.8 +/- 0.7 vs. 7.1 +/- 0.3; p < 0.01).
		Occult cerebral hypoxia is present in the traumatic brain injured patient despite normal traditional measurements of cerebral perfusion.
	www.aast.org /JTrauma_Mar04.html (298 words)

	eMedicine - Altitude Illness - Cerebral Syndromes : Article by Thomas E Dietz, MD (Site not responding. Last check: 2007-11-04)
		Hypoxia is the primary physiological insult on ascent to high altitude.
		The current hypothesis is that hypoxia elicits neurohumeral and hemodynamic responses in the brain that ultimately result in capillary leakage from microvascular beds and edema.
		Factors that might contribute to a hydrostatic brain edema are multiple and include cerebral vasodilation, elevated cerebral capillary pressure, impaired cerebral autoregulation as well as alterations in the permeability of the blood-brain barrier through cytokine activation.
	www.emedicine.com /emerg/topic22.htm (5138 words)

	April 30, 1998-Vol29n30: What's the real cause of mountain sickness?; Research at UB points to leaks in blood-brain ... (Site not responding. Last check: 2007-11-04)
		Research conducted at UB suggests that high-altitude cerebral edema is caused by a disruption in the blood-brain barrier, the specialized layer of cells in cerebral blood vessels that, under normal conditions, allow only certain substances to pass into the brain's circulation.
		Krasney's research using sheep has shown that the symptoms of high-altitude cerebral edema are not caused by an inadequate supply of oxygen or glucose to the brain.
		In addition, Krasney said, it is possible that, in the presence of hypoxia, another chemical messenger, cyclic guanosine monophosphate (cGMP), acts with nitric oxide, a vasodilator, to promote and sustain permeability of the blood-brain barrier.
	www.buffalo.edu /reporter/vol29/vol29n30/n1.html (568 words)

	Cerebral Hypoxia Information Page: National Institute of Neurological Disorders and Stroke (NINDS)
		Cerebral hypoxia refers to a condition in which there is a decrease of oxygen supply to the brain even though there is adequate blood flow.
		When hypoxia lasts for longer periods of time, it can cause coma, seizures, and even brain death.
		The NINDS supports and conducts studies aimed at understanding neurological conditions that can damage the brain, such as cerebral hypoxia.
	www.ninds.nih.gov /disorders/anoxia/anoxia.htm (436 words)

	Cerebral blood flow response to isocapnic hypoxia during slow-wave sleep and wakefulness -- Meadows et al. 97 (4): 1343 ...
		Cerebral blood flow response to isocapnic hypoxia during slow-wave sleep and wakefulness -- Meadows et al.
		that the cerebral vascular response to hypoxia is similarly
		Nocturnal hypoxia and hypercapnia are characteristics of cardiorespiratory
	jap.physiology.org /cgi/content/full/97/4/1343 (3334 words)

	Treatment of hypoxic-ischaemic brain damage by moderate hypothermia -- Edwards et al. 78 (2): 85 -- Archives of Disease ...
		increase in the incidence of cerebral palsy in infants weighing
		Modest hypothermia preserves cerebral energy metabolism during hypoxia-ischemia and correlates with brain damage: A 31P nuclear magnetic resonance study in unanesthetised neonatal rats.
		Cerebral oxidative metabolism during hypothermia and circulatory arrest in newborn dogs.
	fn.bmjjournals.com /cgi/content/full/78/2/F85 (2748 words)

	Nitric oxide mediates hypoxia-induced cerebral vasodilation in humans -- Van Mil et al. 92 (3): 962 -- Journal of ...
		Percent change in cerebral vascular resistance, cerebral blood flow, mean arterial pressure, and heart rate during normoxia.
		Percent change in cerebral vascular resistance, cerebral blood flow, mean arterial pressure, and heart rate during hypoxia.
		hypoxia-induced cerebral vasodilatation is mediated by the release
	jap.physiology.org /cgi/content/full/92/3/962 (3073 words)

	Journal of Cerebral Blood Flow & Metabolism - NMDA Receptor-Dependent Increase of Cerebral Glucose Utilization ... (Site not responding. Last check: 2007-11-04)
		Regional cerebral glucose utilization was assessed by the 2-DG method (Sokoloff et al., 1977), modified for the 7-day-old rat (Vannucci et al., 1989).
		The lumped constant vas estimated from a nomogram for adult rat brain that allows calculation of the lumped constant from the brain and plasma glucose concentrations (Pardrige et al., 1982).
		This has been shown previously and explained in terms of oxidative metabolism of lactate and ketone bodies, because it was not accompanied by any change in cerebral energy utilization, as determined from the changes in the concentrations of glucose, lactate, ATP, and phosphocreatine during 2 min after decapitation (Vannucci et al., 1994).
	www.nature.com /jcbfm/journal/v16/n5/full/9590122a.html (5309 words)

	A template for defining a causal relation between acute intrapartum events and cerebral palsy: international consensus ... (Site not responding. Last check: 2007-11-04)
		Cerebral palsy, which is characterised by non-progressive, abnormal control of movement or posture, is not diagnosed until
		death in the hippocampus, cerebral cortex, and cerebellum.
		that intrapartum hypoxia was not the cause of the cerebral palsy.
	bmj.bmjjournals.com /cgi/content/full/319/7216/1054 (4791 words)

	Histopathologic Correlates of Temporal Diffusion Changes in a Rat Model of Cerebral Hypoxia/Ischemia -- Miyasaka et al. ...
		Early detection of cerebral infarction and hypoxic ischemic encephalopathy in neonates using diffusion-weighted magnetic resonance imaging.
		Early detection of regional cerebral ischemia in cats: comparison of diffusion- and T2-weighted MRI and spectroscopy.
		Cerebral energy metabolism studied with phosphorous NMR spectroscopy in normal and birth-asphyxiated infants.
	www.ajnr.org /cgi/content/full/21/1/60 (3736 words)

	Cerebral Ischemic Hypoxia: Discrepancy between Apparent Diffusion Coefficients and Histologic Changes in Rats -- ...
		Dynamics of cerebral tissue injury and perfusion after temporary hypoxia-ischemia in the rat: evidence for region-specific sensitivity and delayed damage.
		A diffusion-weighted proton magnetic resonance imaging study of cerebral ischemia in the gerbil.
		Comparison of diffusion- and T2-weighted MRI for the early detection of cerebral ischemia and reperfusion in rats.
	radiology.rsnajnls.org /cgi/content/full/215/1/199 (4880 words)

	The MSDS HyperGlossary: Hypoxia
		Hypoxia is a deficiency of oxygen in inspired (inhaled) gases or in arterial blood and/or in the tissues.
		The most common symptom of hypoxia is cyanosis, a bluish cast to the skin, lips and/or fingernails.
		Cerebral hypoxia at the Nation Institute of Neurological Disorders and Stroke.
	www.ilpi.com /msds/ref/hypoxia.html (352 words)

	Cerebral Hypoxia During Cardiopulmonary Bypass: A Magnetic Resonance Imaging Study -- Mutch et al. 64 (3): 695 -- The ...
		Cerebral Hypoxia During Cardiopulmonary Bypass: A Magnetic Resonance Imaging Study -- Mutch et al.
		Cerebral Hypoxia During Cardiopulmonary Bypass: A Magnetic Resonance Imaging Study
		hypoxia and correlated with jugular venous oxygen saturation.
	ats.ctsnetjournals.org /cgi/content/abstract/64/3/695 (552 words)

	Hydrogen Peroxide Production in Leukocytes during Cerebral Hypoxia and Reoxygenation with 100% or 21% Oxygen in Newborn ... (Site not responding. Last check: 2007-11-04)
		in endothelial cells in the cerebral vascular bed (19, 20).
		Hypoxia induced a decrease in forebrain oxygen uptake (CMRO
		Kutzsche S, Kirkeby OJ, Rise IR, Saugstad OD 1999 Effects of hypoxia and reoxygenation with 21% and 100% oxygen on cerebral nitric oxide concentration and microcirculation in newborn piglets.
	www.pedresearch.org /cgi/content/full/49/6/834 (4672 words)

	APStracts 10:0085A, 2003. (Site not responding. Last check: 2007-11-04)
		Hemodilutional anemia is associated with increased cerebral neuronal nitric oxide synthase gene expression.
		Severe hemodilutional anemia may reduce cerebral oxygen delivery, resulting in cerebral tissue hypoxia.«fnc1»1 Increased nitric oxide synthase (NOS) expression has been identified following cerebral hypoxia and may contribute to the compensatory increase in cerebral blood flow (CBF) observed after hypoxia and anemia.
		Hemodilutional anemia caused an acute reduction in Pbr{?O2} and an increase in cerebral cortical nNOS mRNA, supporting a role for nNOS in the physiological response to acute anemia.
	www.uth.tmc.edu /apstracts/2003/jap/February/85a.html (346 words)

	Cerebral Hypoxia-Ischemia Stimulates Cytokine Gene Expression in Perinatal Rats -- Szaflarski et al. 26 (6): 1093 -- ...
		Cerebral Hypoxia-Ischemia Stimulates Cytokine Gene Expression in Perinatal Rats -- Szaflarski et al.
		At 4 hours after hypoxia, in samples from lesioned tissue, IL-1ß hybridization signal is increased markedly; by 24 hours after hypoxia, levels return to normal.
		In two additional groups of samples from animals killed 24 hours after hypoxia (not shown), values were equal bilaterally.
	stroke.ahajournals.org /cgi/content/full/26/6/1093 (5044 words)

Try your search on: Qwika (all wikis)