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Topic: Hypertriglyceridaemia

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	Lipid Abnormalities Associated with ART May Be Different Than That Associated with Risk for Cardiovascular Disease in ...
		Familial combined hyperlipidaemia is caused by overproduction of small VLDL particles in the liver, leading to a parallel increase in apolipoprotein B and is associated with increased cardiovascular risk.
		Hypertriglyceridaemia was associated with stavudine and abacavir treatment in a univariate analysis, but no variable survived in the multivariate model.
		Patients with familial hypertriglyceridaemia show a normal number of large triglyceride-rich VLDL particles, resulting in a high ratio of VLDL-triglycerides to VLDL-apolipoprotein B. Patients with familial combined hyperlipidaemia, where the number of normally sized particles is increased, are characterized by a low ratio.
	www.natap.org /2003/feb/020103_2.htm (2660 words)

	MEDLINE_1996-2006 - Results of the search <page 1>
		Hypertriglyceridaemia is thought to be the aetiology in 3% of patients with acute pancreatitis, often associated with poorly controlled diabetes mellitus or chronic alcohol abuse.
		In 2 patients hypertriglyceridaemia was associated with diabetes mellitus, in one patient with pregnancy and in another with chronic alcohol abuse.
		Hypertriglyceridaemia is a common and under-diagnosed etiology of acute non-biliary pancreatitis.
	www.bireme.br /cgi-bin/wxislind.exe/iah/online/?IsisScript=iah/iah.xis&nextAction=lnk&base=MEDLINE_1996-2006&exprSearch=10499250&indexSearch=UI&lang=i (338 words)

	[38] HIV mutations associated with hypertriglyceridaemia
		The lipodystrophy syndrome of HIV patients is characterized by a progressive syndrome of abnormal body fat distribution accompanied by hypertriglyceridaemia.
		Hypertriglyceridaemia was statistically associated with predicted resistance for three nucleoside reverse transcriptase inhibitors, zidovudine, abacavir, and stavudine.
		This was in contrast to silent mutations, which occurred at equivalent rates in retroviral genotypes of patients with and without hypertriglyceridaemia.
	www.aegis.org /conferences/Lipo/2002/38.html (189 words)

	raised triglyceride - General Practice Notebook
		Hypertriglyceridaemia (particularly with levels > 10 mmol/L) is a risk factor for acute pancreatitis.
		The association between hypertriglyceridaemia and risk of coronary heart disease is complex and not necessarily dependent on triglyceride level.
		In severe hypertriglyceridaemia, even in fasting samples, chylomicrons will be a major contributor to the hyperlipidaemia (both chylomicrons and VLDL) compete for clearance via lipoprotein lipase.
	www.gpnotebook.co.uk /simplepage.cfm?ID=x20021112002036665170 (966 words)

	IngentaConnect Tamoxifen-Induced Severe Hypertriglyceridaemia and Acute Pancreat...
		Hypertriglyceridaemia is a well known risk factor for acute pancreatitis.
		Hypertriglyceridaemia may be primary in origin or secondary to alcohol abuse, diabetes mellitus, pregnancy or use of drugs.
		We report on a patient with tamoxifen-induced acute pancreatitis and hypertriglyceridaemia who was successfully treated with insulin infusion and long-term gemfibrozil.
	www.ingentaconnect.com /content/adis/cdi/2006/00000026/00000005/art00007 (130 words)

	HealthSentinel.com
		There is a statistically significant reduction of breast cancer among women receiving tamoxifen who also used hormone-replacement therapy during the trial: among 390 women on such therapy and allocated to placebo, we found eight cases of breast cancer compared with one case among 362 women allocated to tamoxifen.
		“Hypertriglyceridaemia was not looked for specifically at each follow-up visit, and the study relies on self-reports from patients and physicians and subsequent confirmation from laboratory findings.
		“The excess of hypertriglyceridaemia seen in women receiving tamoxifen in our study appears to be occurring among women in whom a diagnosis of hypertriglyceridaemia has not previously been made.
	www.healthsentinel.com /briefs.php?event=briefs_print_list_item&id=30&title=Tamoxifen (1178 words)

	Food and nutrition bulletin, volume 11, number 1, march 1989
		The observation that hypertriglyceridaemia occurs in patients with AIDS is intriguing and implies a metabolic derangement.
		Hypertriglyceridaemia also did not correlate with hypercholesterolaemia, with ongoing acute complications, or with a wasting course.
		No correlation between the presence of fasting hypertriglyceridaemia and the presence of circulating HIV p24 antigen, a putative marker of HIV production [28], was found in a recent preliminary study from our laboratory, indicating that there is no simple relationship between these parameters.
	www.unu.edu /Unupress/food/8F111e/8F111E09.htm (2923 words)

	Hypertriglyceridaemia and hyperuricaemia are risk factors for progression of IgA nephropathy -- Syrjänen et al. 15 ...
		Hypertriglyceridaemia and hyperuricaemia are risk factors for progression of IgA nephropathy -- Syrjänen et al.
		Hypertriglyceridaemia and hyperuricaemia are risk factors for progression of IgA nephropathy
		The differences in the cumulative proportion of patients with stable renal function between patients with or without hypertriglyceridaemia are statistically significant, P<0.001 and P<0.01 for all patients and for subgroup N respectively.
	ndt.oxfordjournals.org /cgi/content/full/15/1/34 (4301 words)

	RedOrbit - Health - Carbohydrate Diets, Postprandial Hyperlipidaemia, Abdominal Obesity & Asian Indians: A Recipe for ... (Site not responding. Last check: 2007-11-05)
		Another mechanism for accelerated atherosclerosis in subjects with postprandial hypertriglyceridaemia is increase in circulating adhesion molecules8.
		Dyslipidaemia in Asian Indians is usually characterized by hypertriglyceridaemia, low levels of HDL, and high levels of small dense LDL (a cluster of abnormalities termed as atherogenic dyslipidaemia) which frequently occurs in conjunction with PPHL9"12.
		In Asian Indians, postprandial hypertriglyceridaemia in treated patients with type 2 diabetes was principally seen in those consuming high carbohydrate and low fat diets24.
	www.redorbit.com /news/display?id=133100 (2364 words)

	s000918a - Uric Acid Nephropathy (Site not responding. Last check: 2007-11-05)
		Hypertriglyceridaemia and hyperuricaemia are risk factors for progression of IgA nephropathy.
		Hypertriglyceridaemia and hyperuricaemia were significantly more common at the time of renal biopsy in patients with progressive than in those with stable disease.
		In patients with normal renal function at the time of diagnosis initial hypertriglyceridaemia, hyperuricaemia, hypertension and proteinuria were independent risk factors for progression of IgAN in the Cox regression hazard model.
	www.emory.edu /WHSCL/grady/amreport/litsrch00/s000918a.html (3111 words)

	Chronic Pancreatitis In Children
		Hypertriglyceridaemia and hyper parathyroidism though rare reported to have chronic pancreatitis in children.
		In our experience of 20 cases of chronic pancreatitis in children, four patients had hereditary pancreatitis, six had idiopathic, eight had tropical pancreatitis while hypertriglyceridaemia and hyperparathyroidism in one case each.
		Chronic pancreatitis is common in children, but when chronic pancreatitis is diagnosed efforts should be made to identify treatable causes like hypertriglyceridaemia, hyper parathyroidism, pancreas divisum and other congenital anomaly.
	www.bhj.org /journal/2001_4301_jan/sp_76.htm (1026 words)

	Postprandial hypertriglyceridaemia in patients with Tangier disease -- Kolovou et al. 56 (12): 937 -- Journal of ...
		Postprandial hypertriglyceridaemia in patients with Tangier disease -- Kolovou et al.
		hypertriglyceridaemia is considered to be a vascular risk.
		hypertriglyceridaemia is not present in all patients with TD,
	jcp.bmjjournals.com /cgi/content/full/56/12/937 (2710 words)

	EASD: Simvastatin Protects against Endothelial Dysfunction in Type 2 Diabetes (Site not responding. Last check: 2007-11-05)
		According to the researchers, from University of Udine, Azienda Ospedaliera S. Maria della Misericordia, and Morpurgo-Hofman Research Laboratory on Aging, all in Udine, Italy, postprandial hypertriglyceridaemia and hyperglycaemia are considered risk factors for cardiovascular disease and may induce endothelial dysfunction.
		He also pointed out that, according to this study, postprandial hyperglycaemia by itself is a damaging factor, but its effects are compounded by the concomitant presence of hypertriglyceridaemia, which is very common in diabetic patients.
		"Moreover, our study shows that statin treatment counterbalances the effect of both postprandial hyperglycaemia and hypertriglyceridaemia, and that this effect is not related to the lipid lowering effect of the compound," he said.
	www.pslgroup.com /dg/21CF7A.htm (503 words)

	Carbohydrate-induced hypertriglyceridaemia among West Indian diabetic & non diabetic subjects after ingestion of ...
		In this study we therefore aimed to determine which of the three popular carbohydrate foods has the highest potential of increasing postprandial triglyceride levels in type 2 diabetic patients and in healthy non diabetic individuals.
		The link between postprandial hyperglycaemia and CVD risk in type 2 diabetes appears to be related to postprandial hypertriglyceridaemia and the increased levels of highly atherogenic small and dense low density lipoprotein (LDL) particles8-10.
		A recent review of studies on dietary carbohydrate (fat/carbohydrate ratio compositions) that is most likely to induce hypertriglyceridaemia and its net effect on CVD risk brought to focus the central issue of the type of dietary carbohydrate that has the least potential of inducing hypertriglyceridaemia11.
	findarticles.com /p/articles/mi_qa3867/is_200501/ai_n11850184 (748 words)

	Carbohydrate-induced hypertriglyceridaemia among West Indian diabetic & non diabetic subjects after ingestion of ...
		In this study we therefore aimed to determine which of the three popular carbohydrate foods has the highest potential of increasing postprandial triglyceride levels in type 2 diabetic patients and in healthy non diabetic individuals.
		The link between postprandial hyperglycaemia and CVD risk in type 2 diabetes appears to be related to postprandial hypertriglyceridaemia and the increased levels of highly atherogenic small and dense low density lipoprotein (LDL) particles8-10.
		A recent review of studies on dietary carbohydrate (fat/carbohydrate ratio compositions) that is most likely to induce hypertriglyceridaemia and its net effect on CVD risk brought to focus the central issue of the type of dietary carbohydrate that has the least potential of inducing hypertriglyceridaemia11.
	www.findarticles.com /p/articles/mi_qa3867/is_200501/ai_n11850184 (764 words)

	News - Fenofibrate, Losartan Show Additive Benefits with Anti-hyperuricaemic Agents in Gout Patients
		Twenty-seven males (average age 50) with gout and hypertriglyceridaemia were given 300 mg of fenofibrate once daily for 2 months and were simultaneously treated with 50 mg benzbromarone once daily for those with underexcretory gout or with 200 mg allopurinol twice daily for those with overexcretory gout.
		In the hypertension study, 25 males (average age 54) with gout and hypertension were administered 50 mg losartan daily for 2 months in conjunction with the same anti-hyperuricaemic treatment described for the hypertriglyceridaemia study.
		Furthermore, all study groups showed significant decreases in serum uric acid upon supplementation of anti-hyperuricaemic agent with either losartan or fenofibrate treatment, which was most likely due to increased uric acid clearance and urinary excretion.
	www.docguide.com /news/content.nsf/news/8525697700573E1885256D2F00313D13 (645 words)

	European Society of Clinical Microbiology and Infectious Diseases
		Manfredi R. Aim of our study is to assess prospectively the efficacy and safety profile of polyunsaturated fatty acids ethyl ester (PFAEE) in the control of hypertriglyceridaemia complicating antiretroviral-treated HIV disease.
		Forty-three patients aged 38–61 years (29 males) with a diet- and exercise-resistant hyperlipidaemia and a mean triglyceridaemia of 298.3 ± 42.2 mg/dL, received PFAEE at 1 g twice daily, and were followed quartely for at least 1 year.
		For isolated or predominant hypertriglyceridaemia, PFAEE may represent an effective and safe alternative to fibrates and/or statins, to be confirmed in enlarged, randomised, dose-finding comparative trials.
	www.blackwellpublishing.com /eccmid14/abstract.asp?id=14291 (377 words)

	A new drug target for treatment of dyslipidaemia associated with type 2 diabetes and the metabolic syndrome?
		Dyslipidaemia associated with type 2 diabetes and the metabolic syndrome is characterised by moderate hypertriglyceridaemia, low concentrations of HDL cholesterol, cholesterol-rich remnant-lipoprotein particles, and small dense LDL-cholesterol particles.
		Insulin resistance at the adipocyte leads to release of free fatty acids (FFA), and the increased FFA flux to the liver stimulates synthesis and release of VLDL with consequent hypertriglyceridaemia.1 The other source of triglycerides in the circulation are the chylomicrons which carry dietary triglycerides in the postprandial period.
		Hypertriglyceridaemia stimulates lipid exchange (cholesteryl esters and triglycerides) between lipoproteins; this exchange together with the action of various enzymes, such as hepatic lipase, results in the qualitative changes in lipoproteins which are important components of the dyslipidaemia.
	www.malattiemetaboliche.it /articoli/A_new_dug.htm (1095 words)

	Determinants of mild fasting hypertriglyceridaemia in non-insulin-dependent diabetes - Publication - RTI International
		Factors contributing to fasting hypertriglyceridaemia were studied in 20 patients with non-insulin-dependent diabetes--nine with normal triglyceride concentrations [fasting triglyceride 0.94 (range 0.58-1.23) mmol l-1] and eleven with mild fasting hypertriglyceridaemia [fasting triglyceride 2.4 (1.82-4.0) mmol l-1].
		The patients with hypertriglyceridaemia were more obese [body mass index 29.0 (24.6-33.8) vs. 25.7 (21.9-30.1) kg m-2, P less than 0.05] and demonstrated impaired glucose disposal in response to exogenous insulin at isoglycaemia [insulin sensitivity index, SIp 0.7 (0.27-2.5) vs. 2.4 (0.62-5.1) ml m-2 min per mU l-1, P less than 0.001].
		We propose that defects in both the glucoregulatory and antilipolytic actions of insulin contribute to mild fasting hypertriglyceridaemia in NIDDM, and that these defects cannot be attributed solely to obesity.
	www.rti.org /abstract.cfm?pubid=7839 (280 words)

	Mrs. Chand, a thirty year old female presented with history of recurrent attacks of pain... (Site not responding. Last check: 2007-11-05)
		It revealed hypertriglyceridaemia of 1,100 mg% and 1,420 mg% respectively.
		Diagnosis of familial hypertriglyceridaemia (phenotype IV) with recurrent pancreatitis was made.
		Familial hypertriglyceridaemia (type IV) is characterised by high VLDL only.
	www.indegene.com /Gas/ClinRound/indGasCase19.html (409 words)

	BioMed Central \| Full text \| Metabolic risk factor profile associated with use of second generation antipsychotics: a ...
		Hyperlipidaemic effects include both hypertriglyceridaemia and hypercholesterolaemia, although relatively greater effects may be seen in triglyceridaemia [5,6].
		Overweight, hyperglycaemia and hyperlipidaemia are risk factors for cardiovascular events, such as myocardial infarction and stroke, and are especially relevant in patients with schizophrenia because they tend to exhibit other major risk factors such as smoking and a sedentary lifestyle [4,7].
		Moreover the odds of receiving a diagnosis of hypertriglyceridaemia was 4 times significantly higher for patients treated with SGA than for the reference group (OR 4.00, 95%CI 1.39–11.46, p = 0.007); this risk was not affected by adjusting for age (OR 4.00, 95%CI 1.39–11.46, p = 0.007).
	www.biomedcentral.com /1471-244X/6/11 (4929 words)

	Definition — Metabolic Syndrome Institute
		Insulin-resistance is indeed a component of obesity, type 2 diabetes, and in many cases of hypertension, hypertriglyceridaemia with low levels of HDL-cholesterol.
		These abnormalities of lipoprotein metabolism, which are more likely to occur together than separately, and in large part relate to insulin-resistance/ hyperinsulinaemia, have therefore been proposed to constitute key component traits of the Metabolic Syndrome.
		Insulin-resistance favouring the onset of type 2 diabetes develops commonly in association with correlated metabolic abnormalities recognised as cardiovascular risk factors and that are present prior to the onset of diabetes hypertriglyceridaemia, low HDL-cholesterol, increased intra-abdominal fat, hyperinsulinaemia, mild elevation of glycaemia and blood pressure.
	www.metabolic-syndrome-institute.org /medical_information/definition (689 words)

	RedOrbit - Health - Carbohydrate-Induced Hypertriglyceridaemia Among West Indian Diabetic & Non Diabetic Subjects After ... (Site not responding. Last check: 2007-11-05)
		The relatively higher postprandial hypertriglyceridaemia might be related to the fact that 9 of the 10 patients with fasting hypertriglyceridaemia were of East Indian origin In this population, the prevalence of diabetes is higher in people of East Indian origin compared with people of African origin26,27.
		Further the amount of simple sugars contained in each carbohydrate test food was not determined in the present study considering that high simple sugars in carbohydrate diet stimulate fatty acid synthesis and consequently determines the levels of plasma triglyceride after carbohydrate meals29.
		Probably extension of the study duration beyond 2.5 h would have provided a better discrimination between diabetic and non diabetic subjects eventhough 2 h glycaemia after a standard oral glucose tolerance test (75 g glucose) has been shown to be closely related to the level of glycaemia after a standardised meal31.
	www.redorbit.com /news/display?id=133108 (3190 words)

	1 Summary
		The lipoprotein phenotype is most conveniently classified in terms of hypercholesterolaemia, hypertriglyceridaemia or as a mixed picture in which cholesterol and triglyceride levels are more-or-less equally elevated.
		Pure hypertriglyceridaemia is the least atherogenic, but not totally innocuous form of the different profiles, but severe hypertriglyceridaemia can result in acute pancreatitis and other clinically adverse outcomes.
		Severe hypertriglyceridaemia persisting after maximal dietary fat reduction and control of secondary causes may require the combination of a fibrate with niacin or a statin, but such patients should generally be referred to a Lipid Clinic.
	www.lassa.org.za /guidelines/SA_National_Guide.htm (6460 words)

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