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Topic: Polymorphic ventricular tachycardia

Related Topics

Familial long QT syndrome

Dilated cardiomyopathy

Cardiomyopathy

Cardiology

Arrhythmogenic right ventricular dysplasia

Supraventricular tachycardia

Aortic dissection

Electrocardiogram

Torsades de pointes

Tollund Man

Dependent territories of the United Kingdom

Brunel University

Mabinogion

Red Brick university

Branwen

	s000626b - Torsades de Points - Treatment
		Ventricular fibrillation and polymorphic ventricular tachycardia with critical coronary artery stenosis: does bypass surgery suffice?.
		Polymorphic ventricular tachycardia with cardiac sarcoidosis: treatment with low-dose metoprolol and cibenzoline.
		TdP was defined as PVT with a prolonged QT interval.
	www.emory.edu /WHSCL/grady/amreport/litsrch99/s000626b.html (7107 words)

	Ventricular Tachyarrhythmias
		Polymorphic ventricular tachycardia typically is irregular in rate and rhythm and has varying shapes or morphologies on the ECG.
		Ventricular tachycardia may give rise to symptoms such as palpitations, shortness of breath, or lightheadedness, depending upon the rate of the arrhythmia, its duration, and the underlying heart disease.
		Ventricular arrhythmias and syncope are often provoked by increases in heart rate in response to exercise or emotion.
	www.txai.org /edu/irregular/ventricular_tachyarrhythmias.htm (1988 words)

	s000626a - Torsades de Points
		Abstract: Torsades de pointes is a polymorphic form of ventricular tachycardia that is usually associated with prolongation of the QT interval.
		We report a case of QT prolongation and polymorphic ventricular tachycardia (torsades de pointes) that was precipitated by the intravenous administration of erythromycin.
		This ventricular tachyarrhythmia, characterized by rapid (200-250 beats/min) and irregular paroxysms and progressively varying QRS amplitude and polarity, occurred in the absence of electrolyte disturbance, antiarrhythmic drug therapy or acute ischemia.
	www.emory.edu /WHSCL/grady/amreport/litsrch99/s000626a.html (3453 words)

	Welcome to Ventricular Tachycardia
		Ventricular tachycardia is " a tachycardia where the origin of the arrhythmia is within the ventricles".
		That is, the tachycardia does not require the participation of the atrium or AV node and His bundle although the latter structures may be activated "passively" by retrograde conduction.
		Ventricular tachycardia related to acute ischemia or that is extremely rapid is frequently polymorphic, The probability of the presence of VT in a patient with cardiac disease increases in general with the severity of cardiac disease and is most likely to be fatal in patients with impaired ventricular function.
	www.lhsc.on.ca /uwodoc/pages/vt.htm (2014 words)

	Broad Complex Tachycardia - Patient UK
		Ventricular tachycardia is described as either monomorphic (QRS complexes are the same or very similar) or polymorphic (wide beat to beat variation in QRS appearance).
		Ventricular tachycardia is defined as three or more ventricular extrasystoles in succession at a rate of more than 120 beats/min.
		Polymorphic ventricular tachycardia: has the same ECG characteristics as torsades de pointes but in sinus rhythm the QT interval is normal.
	www.patient.co.uk /showdoc/40000558 (1208 words)

	Catecholaminergic polymorphic ventricular tachycardia: electrocardiographic characteristics and optimal therapeutic ...
		A new electrocardiographic entity in the spectrum of idiopathic ventricular tachycardia.
		Ventricular tachycardia and exercise related syncope in children with structurally normal hearts: emphasis on repolarisation abnormality.
		Mutations in the cardiac ryanodine receptor gene (hRyR2) underlie catecholaminergic polymorphic ventricular tachycardia.
	heart.bmjjournals.com /cgi/content/full/89/1/66 (3476 words)

	Ventricular Tachycardia (Site not responding. Last check: 2007-10-24)
		When you have three or more of those complexes in a row, this is called a run of ventricular tachycardia.
		It is important to recognize this arrhythmia because it requires prompt evaluation and treatment, and often heralds impending ventricular fibrillation and/or cardiac arrest.
		Polymorphic ventricular tachycardia is associated with myocardial infarction, acute ischemia, long QT syndrome, and certain drugs.
	campionambulance.com /staff/ventricular_tachycardia.htm (149 words)

	Mayo Clinic Proceedings
		Catecholaminergic polymorphic ventricular tachycardia is another heritable arrhythmia syndrome that typically manifests with exertional syncope or sudden death.
		Catecholaminergic polymorphic ventricular tachycardia is associated with a completely normal resting 12-lead electrocardiogram; CPVT is electrocardiographically suspected after either exercise or catecholamine stress testing that demonstrates significant ventricular ectopy.
		Priori SG, Napolitano C, Tiso N, et al. Mutations in the cardiac ryanodine receptor gene (hRyR2) underlie catecholaminergic polymorphic ventricular tachycardia. Circulation.
	www.mayoclinicproceedings.com /inside.asp?AID=699&UID=2474 (2928 words)

	Polymorphic Ventricular Tachycardia Reviews (Site not responding. Last check: 2007-10-24)
		Most of this 12-lead recording is polymorphic ventricular tachycardia but, in the rhythm strip, the large deflection (arrowed) is the defibrillator discharging.
		Ventricular tachycardia with subtle AV dissociation Torsade de pointes ventricular tachycardia Polymorphic Ventricular Tachycardia with an ICD Ventricular Fibrillation pacemakers Catecholaminergic polymorphic ventricular tachycardia is a rare arrhythmogenic Catecholaminergic polymorphic ventricular tachycardia is an uncommon arrhythmogenic EXPERIMENTAL REVIEWS.
		Polymorphic reentrant ventricular tachycardia in the isolated rabbit heart studied by high for the genesis of polymorphic ventricular tachycardia has recently become emphasized polymorphic ventricular tachycardia I though perfectly Well here's a polymorphic ventricular tachycardia bounce in either in the Unix is surely seen forwarded Message.
	health24hrs.info /cardiology/polymorphic-ventricular-tachycardia.html (563 words)

	Myocardial infarction (Site not responding. Last check: 2007-10-24)
		Ventricular fibrillation - Electrical cardioversion at 200 J, 200- to 300 J, and 300 J, as needed.
		Polymorphic ventricular tachycardia lasting more than 30 seconds or causing hypotension - Electrical cardioversion at 200 J; 200 to 300 J; and 300 J, as needed.
		Treatment of non-sustained ventricular ectopy or accelerated ventricular rhythm.
	www3.kumc.edu /heartdoc/palmguide/ami_acc-05.htm (83 words)

	ABC of clinical electrocardiography: Broad complex tachycardia---Part II -- Edhouse and Morris 324 (7340): 776 -- BMJ (Site not responding. Last check: 2007-10-24)
		ventricular outflow tract, and the impulse spreads inferiorly.
		Ventricular tachycardia and supraventricular tachycardia with bundle branch block may produce similar electrocardiograms.
		The emergency management of a broad complex tachycardia depends on the wellbeing of the patient and the origin of the arrhythmia.
	bmj.bmjjournals.com /cgi/content/full/324/7340/776 (1327 words)

	Inducible polymorphic ventricular tachyarrhythmias in a transgenic mouse model with a long Q-T phenotype -- Jeron et ...
		The tachycardia stops spontaneously after 6 s and is followed by a brief phase of bradycardia, until the regular heart rate is reestablished.
		mice, whereas ventricular tachycardia was not inducible in WT FVB mice.
		Polymorphic ventricular tachycardia cannot be induced in control mice.
	ajpheart.physiology.org /cgi/content/full/278/6/H1891 (4294 words)

	Ventricular Tachycardia After Amiodarone: Report of an Unusual Case
		Amiodarone has been approved for treatment of patients with ventricular and supraventricular tachyarrhythmias.1—3 Previous reports have found that it is effective for the conversion of atrial fibrillation and subsequent maintenance of sinus rhythm.2—6 In this report, we describe a case of atrial fibrillation that converted with amiodarone.
		Although that is a well-documented complication of this drug, this case report underscores the need to remain vigilant for serious and life-threatening arrhythmias in patients who are exposed to amiodarone.
		No additional ventricular tachycardia or torsade de pointes was noted after isoproterenol infusion.
	www.jrnlappliedresearch.com /articles/Vol3Iss2/Tak.htm (1379 words)

	Ventricular Tachycardia - Page 2
		Ventricular tachycardia is an abnormal condition in which the lower
		During ventricular tachycardia, however, additional signals arise from the ventricles.
		Ventricular tachycardias are classified by their duration (e.g., sustained or nonsustained) and whether they occur without cause or because of an underlying condition, such as
	heart.healthcentersonline.com /arrhythmia/VentricularTachycardia2.cfm (400 words)

	Evaluation of Ibutilide Fumarate (Corvert®) (Site not responding. Last check: 2007-10-24)
		The development of polymorphic ventricular tachycardia in ibutilide patients is significantly correlated with female gender, non-White race, presence of heart failure and lower pulse rate.
		Torsades de pointes, a polymorphic ventricular tachycardia that develops in the setting of a prolonged QT interval, may occur because of the direct effect of ibutilide on cardiac repolarization, however, ibutilide can also cause polymorphic ventricular tachycardia in the absence of excessive prolongation of the QT interval.
		Ibutilide infusion should be stopped as soon as the presenting arrhythmia is terminated or in the event of sustained or nonsustained ventricular tachycardia, or marked prolongation of QT or QTc.
	daccx.bsd.uchicago.edu /drug/Bulletins/N0898.html (3334 words)

	Brugada Sydrome - Electrophysiologic and Hemodynamic Findings
		All published studies agree on the inducibility of polymorphic ventricular tachycardia by programmed electrical stimulation in symptomatic patients.
		The induced arrhythmia is sustained in practically all cases, results in hemodynamic collapse and has to be terminated by an external DC shock.
		It can be criticized that polymorphic ventricular tachycardia or ventricular fibrillation induced by programmed stimulation is a non-specific finding, because these arrhythmias can sometimes be induced in patients with a normal heart.
	www.brugada.org /about/disease-electro-hemodynamic.html (230 words)

	Australian Prescriber - Drugs and the QTc interval
		This effect is important as it is associated with polymorphic ventricular tachycardia and possible sudden cardiac death.
		The arrhythmia is usually self-terminating, but can degenerate into ventricular fibrillation or rarely sustained ventricular tachycardia.
		Brief episodes of self-terminating polymorphic ventricular tachycardia do not require any specific treatment apart from withdrawal of the suspect drug and correction of metabolic abnormalities.
	www.australianprescriber.com /magazine/25/3/63/5 (1411 words)

	eMedicine - Torsade de Pointes : Article by Jatin Dave, MD, MPH
		Torsade should be differentiated from polymorphic ventricular tachycardia, monomorphic ventricular tachycardia, and supraventricular tachycardia with aberrant conduction.
		The diagnosis of torsade should be considered in any patient with pause-dependent ventricular tachycardia, and ventricular bigeminy in a patient with long QT interval may be a sign of an impending torsade.
		Nguyen PT, Scheinman MM, Seger J: Polymorphous ventricular tachycardia: clinical characterization, therapy, and the QT interval.
	www.emedicine.com /med/topic2286.htm (3963 words)

	Electrophysiologic Mechanisms of the Long QT Interval Syndromes and Torsade de Pointes -- Tan et al. 122 (9): 701 -- ...
		This beat is seen on the surface electrocardiogram as a premature ventricular depolarization arising from the end of a prolonged QT interval (or in the midst of a pronounced U wave) (dotted line).
		Polymorphous ventricular tachycardia associated with a marked prolongation of the Q-T interval induced by amiodarone.
		Ventricular tachyarrhythmias related to early afterdepolarizations and triggered firing: relationship to QT interval prolongation and potential therapeutic role for calcium channel blocking agents.
	www.annals.org /cgi/content/full/122/9/701 (7720 words)

	Arrhythmic disorder mapped to chromosome 1q42-q43 causes malignant polymorphic ventricular tachycardia in structurally ...
		polymorphic ventricular tachycardia and syncope in the absence
		Identification of patients with ventricular tachycardia after myocardial infarction from signals in the terminal QRS complex.
		Juvenile sudden death and effort ventricular tachycardias in a family with right ventricular cardiomyopathy.
	content.onlinejacc.org /cgi/content/full/34/7/2035 (4366 words)

	Screening for ryanodine receptor type 2 mutations in families with effort-induced polymorphic ventricular arrhythmias ... (Site not responding. Last check: 2007-10-24)
		Screening for ryanodine receptor type 2 mutations in families with effort-induced polymorphic ventricular arrhythmias and sudden death: Early diagnosis of asymptomatic carriers -- Bauce et al.
		Figure 3 Distribution of patients with effort-induced polymorphic ventricular arrhythmias and/or syncope in relation to age.
		Arrhythmic disorder mapped to chromosome 1q42-q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts.
	content.onlinejacc.org /cgi/content/full/40/2/341 (3790 words)

	Absence of Calsequestrin 2 Causes Severe Forms of Catecholaminergic Polymorphic Ventricular Tachycardia -- Postma et ...
		of 100 bpm, preceded by isolated polymorphic ventricular bigeminy.
		Examples of polymorphic VPBs at the beginning of the stress test (B), followed by self-terminating polymorphic salvos as the heart rate increases >100 bpm (C).
		a PVT threshold of 110 bpm, and a QTc interval of 420 ms.
	circres.ahajournals.org /cgi/content/full/91/8/e21 (4033 words)

	Brugada Sydrome - Definition of the Disease (Site not responding. Last check: 2007-10-24)
		The episodes of syncope and sudden death (aborted or not) are caused by fast polymorphic ventricular tachycardias or ventricular fibrillation (picture 2).
		Only in very few cases there is alternation of long-short sequences before the polymorphic ventricular tachycardia, a finding which is very common in other arrhythmias like "torsade de pointes" in the long QT syndrome.
		There is no preceding acceleration in the heart rate as is the case of cathecolamine-dependent polymorphic ventricular tachycardia
	www.brugada.org /about/disease-definition.html (136 words)

	[No title]
		The drug should be given in a setting of continuous EKG monitoring and by personnel trained in treating arrhythmias particularly polymorphic ventricular tachycardia.
		Patients with chronic atrial fibrillation may not be the best candidates for ibutilide since they often revert after conversion and the risks of treatment may not be justified when compared to alternative management.
		placebo), idioventricular rhythm (0.2%), sustained monomorphic ventricular tachycardia (0.2%), renal failure (0.3%) Postmarketing and/or case reports: Erythematous bullous lesions Drug Interactions Antiarrhythmics: Class Ia antiarrhythmic drugs (disopyramide, quinidine, and procainamide) and other class III drugs such as amiodarone and sotalol, should not be given concomitantly with ibutilide due to their potential to prolong refractoriness.
	www.musc.edu /pharmacyservices/Drugs/I/Ibutilide.doc (322 words)

	eMedicine - Torsades de Pointes : Article Excerpt by: Syed Nazeer, MD
		Background: Torsades de pointes, literally meaning twisting of points, is a distinctive form of polymorphic ventricular tachycardia (VT) characterized by a gradual change in the amplitude and twisting of the QRS complexes around the isoelectric line.
		Torsades de pointes (torsades) is associated with a prolonged QT interval, which may be congenital or acquired.
		It usually terminates spontaneously but frequently recurs and may degenerate into sustained VT and ventricular fibrillation.
	www.emedicine.com /med/byname/torsades-de-pointes.htm (611 words)

	Pseudo-ventricular tachycardia: electrocardiographic artefact mimicking non-sustained polymorphic ventricular ...
		Pseudo-ventricular tachycardia: electrocardiographic artefact mimicking non-sustained polymorphic ventricular tachycardia in a patient evaluated for syncope -- Vereckei 90 (1): 81 -- Heart
		Pseudo-ventricular tachycardia: electrocardiographic artefact mimicking non-sustained polymorphic ventricular tachycardia in a patient evaluated for syncope
		and might be assumed to be initiated by a premature ventricular
	heart.bmjjournals.com /cgi/content/full/90/1/81 (283 words)

	Denaturing HPLC-Based Approach for Detecting RYR2 Mutations Involved in Malignant Arrhythmias -- Bagattin et al. 50 ...
		Screening for ryanodine receptor type 2 mutations in families with effort-induced polymorphic ventricular arrhythmia and sudden death: early diagnosis of asymptomatic carriers.
		Clinical and molecular characterization of patients with catecholaminergic polymorphic ventricular tachycardia.
		Laitinen PJ, Swan H, Kontula K. Molecular genetics of exercise-induced polymorphic ventricular tachycardia: identification of three novel cardiac ryanodine receptor mutations and two common calsequestrin 2 amino-acid polymorphisms.
	www.clinchem.org /cgi/content/full/50/7/1148 (3888 words)

	LQTS (Site not responding. Last check: 2007-10-24)
		Ryanodine receptor mutations associated with stress-induced ventricular tachycardia mediate increased calcium release in stimulated cardiomyocytes.
		Bidirectional ventricular tachycardia and fibrillation elicited in a knock-in mouse model carrier of a mutation in the cardiac ryanodine receptor.
		Absence of calsequestrin 2 causes severe forms of catecholaminergic polymorphic ventricular tachycardia.
	www.sfu.ca /~tibbits/courses/K812/CPVT.htm (158 words)

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